Intestinal amoebiasis granulomatous with appendicitis

Amoeba intestinal lesion mainly in the cecum, ascending colon, rectum and sigmoid colon followed by a serious case of the entire colon and small intestine may be involved in the following paragraph. For the diseases associated with the organization dissolved liquefaction necrosis of the inflammation, can be divided into acute phase and chronic phase. The naked eye view of the acute stage disease, early in the mucosal surface can be seen most of the uplift of the sallow cap the size of the needle point or necrosis in shallow ulcers. Progress of disease, increasing necrotizing lesions, were round-shaped button, there is bleeding around with wrapping. At this point in the mucosal layer trophozoites continue to breed destruction, through the mucous membrane and muscle submucosal reach. As the submucosa loose organization, easy-to-amoeba to spread around, fell off after the liquefied necrotic tissue to form a small port at the end of the large flask-shaped ulcer, was at risk of sneak (undermined), with a diagnosis of the disease significance. Mucosal ulceration between the performance of normal or only mild catarrhal inflammation. In severe cases, adjacent mucosa ulcer in the lower tunnel-like form to communicate with each other, the mucosal surface can be large pieces of necrosis off to form the edge of the great sneak ulcers, which can reach a diameter of 8 ~ 12cm. Clinically, the acute stage for intestinal symptoms, for the performance of abdominal pain, diarrhea, stool containing mucus as a result of increased volume and a large amount of blood and necrosis of the intestinal wall dissolved the organization and was purple or dark red paste, with Xingchou. Stool examination when the amoeba trophozoites easy to find. As the disease rectum and anus disease less, so as bacillary dysentery symptoms Lijihouzhong clear that the general performance of the poisoning is very minor. Most of the acute stage curable. A small number of cases, the ulcer too, which can cause intestinal perforation. However, this disease develop a more moderate disease, perforated ulcer before the end of the regular serosa layer adhesion with neighboring organizations, it is only perforated to form the limitations of abscess, caused little diffuse peritonitis. Wall of the small blood vessels rupture caused by bleeding were more common, but the blood vessels leading to massive destruction of those who are very rare. As a result of inadequate treatment of a small number of timely, thorough and transferred to the chronic phase. Chronic diseases are complex diseases. Some have ulcer healing, while others ulcer can continue to exist and expand, and even has healing necrotic ulcers occur again. Necrosis, ulcers, proliferation of granulation tissue and scar formation both at the same time, the proliferation of mucous membrane can be formed polyps, which will ultimately enable the normal form of intestinal completely lost. Wall can be fibrous tissue hyperplasia and thickening and hardening, and even cause intestinal stenosis. In some cases may be due to excessive proliferation of granulation tissue, and the formation of the limitations of mass, known as the swollen amoeba (amoeboma), was particularly prevalent in the cecum, appendix, said amoeba. Clinically misdiagnosed as colon cancer.